AntiAge Cancer Research Results
AntiAge, Anti-aging: Click to Expand ⟱
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Anti-aging strategies hold promise for improving overall health and potentially reducing cancer risk, the intricate interplay between the molecular mechanisms of aging and cancer requires careful consideration.
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Nor, Normal Healthy: Click to Expand ⟱
Scientific Papers found: Click to Expand⟱
*CRM↓, *Dose?, *AntiAge↑, *Acetyl-CoA↓, *SIRT1↑, *AMPK↑, *mTORC1↓, *AntiAge↑, chemoP↑,
*OS↑, *Inflam↓, *LDL↓, *AntiAge↑, *GSH↑, *SOD↑, *AMPKα↑, *Weight∅,
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vitro+vivo, |
Nor, |
HUVECs |
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*MMP↑, *Hif1a↑, *SIRT1↑, *ROS↓, *AntiAge↑, *Dose↝, *angioG↑,
eff↑, *antiOx↑, *MMP9↓, *TNF-α↓, *NF-kB↓, *SOD↑, *Catalase↑, *AntiAge↑, *Inflam↓, *JNK↓,
*ROS↓, *antiOx↑, *AntiAge↑, *MMP↑, *ECAR↓, *Glycolysis↓, *PAR-2↓, *CXCL12↑, *BMAL1↑, *mt-ROS↓, *OXPHOS↓,
*AntiDiabetic↑, *AntiAge↑, *toxicity⇅, *CRM↑, *Strength↑, *LDL↓, *AMPK↑, *TAC↑, *ROS↓, *Inflam↓, Risk↓, *cardioP↑, *ALAT↓, *NRF2↑, *SOD2↑, *TrxR1↑, *NQO1↑, *NQO2↑,
*AntiAge↑, *AMPK↑, *mPGES-1↓, *Ca+2↑, *ER Stress↑, *OS↑, *ROS↓,
*Dose↝, *AntiAge↑,
*AntiAge↑, *eff↑, *Inflam↓, *AntiCan↑, *ROS↓, *Catalase↑, *GSR↑, *HO-1↑, *NAD↑, *NQO1↑, *SOD↑, *NRF2↑,
*AntiAge↑, *IGF-1↓, *AMPK↑, *CRM↑, *PGC-1α↑, *mtDam↓, *motorD↑, *hepatoP↑, *Dose↝,
*AntiAge↑, *Dose↑, *BioAv↑, *BioAv↑, *BioAv∅, *BioAv↑, *BioAv↑, *BioAv↑, *BioAv↑,
*RenoP↑, *Inflam↓, *NRF2↑, *HO-1↑, *SIRT1↑, *ROS↓, AntiAge↑,
*antiOx↑, *Inflam↓, *AntiAge↑, *SIRT1↑, *SIRT3↑, *FOXO↑, *ROS↓,
*EP300↓, *mTORC1↓, *CRM↑, *HATs↓, *p62↓, *AntiAge↑, AntiCan↑,
cardioP↑, eff↓, AntiAge↑, BioAv↑, CRM↝,
*AntiCan↑, *Inflam↓, *antiOx↑, *AntiAge↑, *hepatoP↑, *cardioP↑, *neuroP↑, *eff↑,
Showing Research Papers: 1 to 16 of 16
* indicates research on normal cells as opposed to diseased cells
Total Research Paper Matches: 16
Pathway results for Effect on Cancer / Diseased Cells:
Core Metabolism/Glycolysis ⓘ
CRM↝, 1,
Drug Metabolism & Resistance ⓘ
BioAv↑, 1, eff↓, 1, eff↑, 1,
Functional Outcomes ⓘ
AntiAge↑, 2, AntiCan↑, 1, cardioP↑, 1, chemoP↑, 1, Risk↓, 1,
Total Targets: 9
Pathway results for Effect on Normal Cells:
Redox & Oxidative Stress ⓘ
antiOx↑, 4, Catalase↑, 2, GSH↑, 1, GSR↑, 1, HO-1↑, 2, NQO1↑, 2, NRF2↑, 3, OXPHOS↓, 1, ROS↓, 7, mt-ROS↓, 1, SIRT3↑, 1, SOD↑, 3, SOD2↑, 1, TAC↑, 1, TrxR1↑, 1,
Mitochondria & Bioenergetics ⓘ
MMP↑, 2, mtDam↓, 1, PGC-1α↑, 1,
Core Metabolism/Glycolysis ⓘ
Acetyl-CoA↓, 1, ALAT↓, 1, AMPK↑, 4, BMAL1↑, 1, CRM↓, 1, CRM↑, 3, ECAR↓, 1, Glycolysis↓, 1, LDL↓, 2, NAD↑, 1, SIRT1↑, 4,
Cell Death ⓘ
JNK↓, 1,
Kinase & Signal Transduction ⓘ
AMPKα↑, 1,
Transcription & Epigenetics ⓘ
HATs↓, 1,
Protein Folding & ER Stress ⓘ
ER Stress↑, 1, NQO2↑, 1,
Autophagy & Lysosomes ⓘ
p62↓, 1,
Proliferation, Differentiation & Cell State ⓘ
EP300↓, 1, FOXO↑, 1, IGF-1↓, 1, mTORC1↓, 2,
Migration ⓘ
Ca+2↑, 1, CXCL12↑, 1, MMP9↓, 1,
Angiogenesis & Vasculature ⓘ
angioG↑, 1, Hif1a↑, 1,
Immune & Inflammatory Signaling ⓘ
Inflam↓, 7, mPGES-1↓, 1, NF-kB↓, 1, PAR-2↓, 1, TNF-α↓, 1,
Drug Metabolism & Resistance ⓘ
BioAv↑, 6, BioAv∅, 1, Dose?, 1, Dose↑, 1, Dose↝, 3, eff↑, 2,
Clinical Biomarkers ⓘ
ALAT↓, 1,
Functional Outcomes ⓘ
AntiAge↑, 15, AntiCan↑, 2, AntiDiabetic↑, 1, cardioP↑, 2, hepatoP↑, 2, motorD↑, 1, neuroP↑, 1, OS↑, 2, RenoP↑, 1, Strength↑, 1, toxicity⇅, 1, Weight∅, 1,
Total Targets: 68
Scientific Paper Hit Count for: AntiAge, Anti-aging
Query results interpretion may depend on "conditions" listed in the research papers.
Such Conditions may include :
-low or high Dose
-format for product, such as nano of lipid formations
-different cell line effects
-synergies with other products
-if effect was for normal or cancerous cells
Filter Conditions: Pro/AntiFlg:% IllCat:% CanType:49 Cells:% prod#:% Target#:1245 State#:% Dir#:2
wNotes=0 sortOrder:rid,rpid
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